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Researchers Identify New Genes Linked With Schizophrenia Risk in First-of-Its-Kind Study

Researchers have made an important discovery about the causes of schizophrenia, a disease related to dementia or fragmented mentality.   Researchers have identified two genes associated with the disease as well as a third gene that carries the risk of schizophrenia and autism. Scientists involved in this research believes, this discovery can go a long way in finding a cure for these kind of diseases. Researchers at the Icahn School of Medicine found that these harmful genes are almost the same in every ethnic or racial group. The findings of this research were published in Nature Genetics . According to an estimate, about one percent of people worldwide suffering from schizophrenia. The scientists identified two risky genes, SRRM2 and AKAP11, based on a comparative analysis of gene sequencing from individuals with schizophrenia and healthy individuals. It compared a dataset of 35,828 patients with schizophrenia to 107,877 healthy or control groups and included a variety of

HIV is Not Eradicated From the Root Due to Lack of CD-73 Protein

Antiretroviral therapy is known to limit the replication of the Human Immunodeficiency Virus (HIV) and destroy infected cells.

But this virus is never completely eradicated from the human body. It retains its presence in some cells and goes deep into the tissues. HIV is a far cry from the human immune system.

Researchers at the University of Alberta in Canada have discovered why infected people cannot get rid of HIV/AIDS. The team found that the T-cells of HIV patients had very low amounts of a protein called CD-73.

T-cells are a type of white blood cell responsible for recognizing and destroying infected cells.

University immunologist Shokrola Elahi concluded the study, published in the journal PLOS Pathogens, "Since CD-73 is responsible for the transfer and movement of cells in tissue, the lack of the protein causes T-cells to detect and detect HIV-infected cells." weak in destroying them.

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