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Researchers Identify New Genes Linked With Schizophrenia Risk in First-of-Its-Kind Study

Researchers have made an important discovery about the causes of schizophrenia, a disease related to dementia or fragmented mentality.   Researchers have identified two genes associated with the disease as well as a third gene that carries the risk of schizophrenia and autism. Scientists involved in this research believes, this discovery can go a long way in finding a cure for these kind of diseases. Researchers at the Icahn School of Medicine found that these harmful genes are almost the same in every ethnic or racial group. The findings of this research were published in Nature Genetics . According to an estimate, about one percent of people worldwide suffering from schizophrenia. The scientists identified two risky genes, SRRM2 and AKAP11, based on a comparative analysis of gene sequencing from individuals with schizophrenia and healthy individuals. It compared a dataset of 35,828 patients with schizophrenia to 107,877 healthy or control groups and included a variety of
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Insulin Deficiency From Insulin Gene Mutation Leads to Smaller Pancreas

A new thing has come to light about the case of insulin deficient diabetes type-1 patients. Researchers found that the pancreas of such patients is smaller than the pancreas of a healthy person.   Beta cells responsible for producing insulin make up a very small portion of the pancreas. Therefore, it was not expected to reduce the size of pancreas due to their degradation in type-1 diabetes. Researchers at Vanderbilt University Medical Center studied to a family living in Alabama and found that insulin deficiency, not autoimmune diabetes, is the primary cause of pancreatic shortening in type 1 diabetes. Four out of eight members of this family were suffering from monogenetic diabetes. It is caused by a rare mutation in the insulin gene.   This leads to insulin deficiency without autoimmunity. MRI of pancreas found that it was smaller in size and abnormal in shape in diabetic patients. This was similar to what was observed in individuals with type 1 diabetes.  The research
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Stone Age Humans Wore Bear Skins 300,000 Years Ago

How did prehistoric humans protect himself from adverse weather impacts?  We have very little information about it. Recently, archaeologists from Germany have claimed that contemporary humans probably used bear skins to cover their bodies.  Bear claws and other body parts remains found in a cave suggest that bears and other fur-bearing animals were hunted for their fur about three million years ago. Author Ivo Verheijen from the University of Tumbingen in Germany says that some prehistoric sites show evidence of bear skinning. Germany's Schöningen is important in such places. According to the study, fur, leather, and other organic materials cannot be preserved for more than a million years, so there is very little direct evidence of clothing. Bears' hair was windproof According to the researchers, the size of the prehistoric bears was very large, their size was as big as the polar bears which became extinct about 25000 years ago.   Those bears had long hair on t
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A New Type of Human Lung Cell Discovered

Recently, new information has emerged about lung diseases. Scientists have identified a new type of cell in the depths of the human lung. According to him the newly discovered cell plays an important role in lung diseases. This research was published in the science journal ' Nature '. The researchers did a thorough analysis of human lung tissue to identify these cells. He named these cells Respiratory Airway Secretory Cells (RASCS). These airway branching cells are located deep in the lungs near the alveoli structure, where the exchange of carbon dioxide and oxygen takes place. Scientists found that RASCS has a kind of stem cell-like properties. This enables them to regenerate cells that are necessary for the normal functioning of the alveoli. Researchers have also found evidence that chronic obstructive pulmonary disease (COPD) caused by smoking interferes with the rebirth process of RASCS. This suggests that blocking this barrier may be a better approach to COPD t
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HIV is Not Eradicated From the Root Due to Lack of CD-73 Protein

Antiretroviral therapy is known to limit the replication of the Human Immunodeficiency Virus (HIV) and destroy infected cells. But this virus is never completely eradicated from the human body. It retains its presence in some cells and goes deep into the tissues. HIV is a far cry from the human immune system. Researchers at the University of Alberta in Canada have discovered why infected people cannot get rid of HIV/AIDS. The team found that the T-cells of HIV patients had very low amounts of a protein called CD-73. T-cells are a type of white blood cell responsible for recognizing and destroying infected cells. University immunologist Shokrola Elahi concluded the study, published in the journal PLOS Pathogens, "Since CD-73 is responsible for the transfer and movement of cells in tissue, the lack of the protein causes T-cells to detect and detect HIV-infected cells." weak in destroying them.
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